The Role of Mitochondrial Biogenesis in Ischemic Stroke

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dc.authorscopusid57216431373
dc.authorscopusid57212299393
dc.authorscopusid57221716042
dc.authorscopusid58531448400
dc.authorscopusid57209599405
dc.authorscopusid58531746400
dc.authorscopusid55565668500
dc.contributor.authorShademan, B.
dc.contributor.authorAvcı, C.B.
dc.contributor.authorKaramad, V.
dc.contributor.authorSoureh, G.J.
dc.contributor.authorOlia, J.B.H.
dc.contributor.authorEsmaily, F.
dc.contributor.authorNourazarian, A.
dc.date.accessioned2024-08-25T18:32:44Z
dc.date.available2024-08-25T18:32:44Z
dc.date.issued2023
dc.departmentEge Üniversitesien_US
dc.description.abstractIschaemic stroke is a sudden neurological disorder caused by localised cerebral ischaemia and persistent cerebral infarction. Occlusion of large arteries due to atherothrombosis, cerebral embolism (i.e., embolic infarction), no thrombotic occlusion in small, deep cerebral arteries (i.e., lacunar infarction), and stenosis of proximal arteries due to hypotension leading to decreased cerebral blood flow in arterial supply zones are the most common causes of ischemic stroke (i.e., hemodynamic stroke). It is now known that organelles play an important role in various signaling events and cellular functions. The molecular mechanisms of mitochondria are involved in cerebral ischemia by generating and scavenging reactive oxygen species, apoptosis, biogenesis, mitochondrial dynamics, and inflammation are all examples of electron transport chain dysfunction. More knowledge about the involvement of mitochondria in ischemia-induced neuronal death and neuronal protection will contribute to the development of better treatment programs for stroke syndromes such as ischemic stroke. © 2023 The Author(s). Published by IMR Press.en_US
dc.identifier.doi10.31083/j.jin2204088
dc.identifier.issn0219-6352
dc.identifier.issue4en_US
dc.identifier.pmid37519159en_US
dc.identifier.scopus2-s2.0-85167569103en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.urihttps://doi.org/10.31083/j.jin2204088
dc.identifier.urihttps://hdl.handle.net/11454/100324
dc.identifier.volume22en_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherIMR Press Limiteden_US
dc.relation.ispartofJournal of Integrative Neuroscienceen_US
dc.relation.publicationcategoryDiğeren_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.snmz20240825_Gen_US
dc.subjectelectron transport chainen_US
dc.subjectischemic strokeen_US
dc.subjectmitochondrial biogenesisen_US
dc.subjectneuronal deathen_US
dc.subjectreactive oxygen metaboliteen_US
dc.subjectapoptosisen_US
dc.subjectbrain mitochondrionen_US
dc.subjectcell functionen_US
dc.subjectcell structureen_US
dc.subjecthumanen_US
dc.subjectischemic strokeen_US
dc.subjectmitochondrial biogenesisen_US
dc.subjectmitochondrial dynamicsen_US
dc.subjectmitochondrial respirationen_US
dc.subjectnerve cellen_US
dc.subjectnerve degenerationen_US
dc.subjectnervous system inflammationen_US
dc.subjectnonhumanen_US
dc.subjectoxidative stressen_US
dc.subjectpathophysiologyen_US
dc.subjectrespiratory chainen_US
dc.subjectReviewen_US
dc.titleThe Role of Mitochondrial Biogenesis in Ischemic Strokeen_US
dc.typeReview Articleen_US

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