Bilateral thalamic infarction - Clinical, etiological and MRI correlates

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dc.contributor.authorKumral, E
dc.contributor.authorEvyapan, D
dc.contributor.authorBalkir, K
dc.contributor.authorKutluhan, S
dc.date.accessioned2019-10-27T18:21:08Z
dc.date.available2019-10-27T18:21:08Z
dc.date.issued2001
dc.departmentEge Üniversitesien_US
dc.description.abstractTo determine clinical, behavioral, topographic and etiological patterns in patients with simultaneous bilateral thalamic infarction in varied thalamic artery territories, we studied 16 patients who were admitted to our stroke unit over a 7-year period. Patients with bithalamic infarction represented 0.6% of our registry which included 2750 ischaemic stroke patients. On computed tomography and magnetic resonance imaging with gadolinium enhancement, there were 4 topographic patterns of infarction: 1) bilateral infarcts in the territory of paramedian artery (8 patients [50%]); 2) bilateral infarcts in the territory of thalamogeniculate arteries (3 patients [19%]); 3) bilateral infarcts involving territory of paramedian and thalamogeniculate arteries (3 patients [19%]); 4) bilateral infarcts involving territory of polar and thalamogeniculate arteries (2 patients [13%]). A specific clinical picture was found in up to 50% of the patients with bithalamic infarction. This included patients with bilateral paramedian infarction having disorder of consciousness, memory dysfunctions, various types of vertical gaze palsy and psychic changes. Bilateral sensory loss predicted accurately bilateral infarction in the territory of thalamogeniculate arteries. The main cause of bilateral thalamic infarction was small artery-disease, followed by cardioembolism. Cognitive functions in patients with bilateral paramedian infarction did not change significantly during the follow-up, in contrast to those with infarcts in varied arterial territories. Acute bilateral infarction involving both thalamus is uncommon, although they are often associated with specific neurologic-neuropsychological patterns, allowing diagnosis before radiological examination.en_US
dc.identifier.doi10.1034/j.1600-0404.2001.00141.xen_US
dc.identifier.endpage42en_US
dc.identifier.issn0001-6314
dc.identifier.issn1600-0404
dc.identifier.issue1en_US
dc.identifier.pmid11153886en_US
dc.identifier.startpage35en_US
dc.identifier.urihttps://doi.org/10.1034/j.1600-0404.2001.00141.x
dc.identifier.urihttps://hdl.handle.net/11454/35861
dc.identifier.volume103en_US
dc.identifier.wosWOS:000166096000007en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherWileyen_US
dc.relation.ispartofActa Neurologica Scandinavicaen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectischaemic strokeen_US
dc.subjectthalamusen_US
dc.subjectdementiaen_US
dc.titleBilateral thalamic infarction - Clinical, etiological and MRI correlatesen_US
dc.typeArticleen_US

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