Studies of the development of congenital anomalies in embryos of riboflavin-deficient, galactoflavin fed rats. II. Role of the terminal electron transport systems

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dc.contributor.authorAksu O.
dc.contributor.authorMackler B.
dc.contributor.authorShepard T.H.
dc.contributor.authorLemire R.J.
dc.date.accessioned2019-10-27T08:00:21Z
dc.date.available2019-10-27T08:00:21Z
dc.date.issued1968
dc.departmentEge Üniversitesien_US
dc.description.abstractStudies of the tissues of embryos and fetuses of rats treated with a riboflavin-deficient, galactoflavin-augmented diet during pregnancy were performed, and demonstrated that the activities of the succinic and DPNH oxidase systems (the terminal electron transport systems) were markedly reduced from control values during the critical period of development (days 12 thru 16 of gestation). The activities of the electron transport systems of placental tissue and maternal heart tissue were unaffected during the same period, suggesting that fetal tissue is particularly susceptible to the effects of riboflavin deficiency. Copyright © 1968 Wiley-Liss, Inc., A Wiley Companyen_US
dc.identifier.doi10.1002/tera.1420010110en_US
dc.identifier.endpage102en_US
dc.identifier.issn0040-3709
dc.identifier.issue1en_US
dc.identifier.pmid4302703en_US
dc.identifier.scopusqualityN/Aen_US
dc.identifier.startpage93en_US
dc.identifier.urihttps://doi.org/10.1002/tera.1420010110
dc.identifier.urihttps://hdl.handle.net/11454/24697
dc.identifier.volume1en_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.relation.ispartofTeratologyen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.titleStudies of the development of congenital anomalies in embryos of riboflavin-deficient, galactoflavin fed rats. II. Role of the terminal electron transport systemsen_US
dc.typeArticleen_US

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