Ponatinib and STAT5 Inhibitor Pimozide Combined Synergistic Treatment Applications Potentially Overcome Drug Resistance via Regulating the Cytokine Expressional Network in Chronic Myeloid Leukemia Cells

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dc.authoridTezcanli Kaymaz, Burcin/0000-0003-1832-1454
dc.authoridCelik, Besne/0000-0001-5417-8005
dc.contributor.authorTezcanli Kaymaz, Burcin
dc.contributor.authorGumus, Nurcan
dc.contributor.authorCelik, Besne
dc.contributor.authorAlcitepe, Ilayda
dc.contributor.authorBiray Avci, Cigir
dc.contributor.authorAktan, Cagdas
dc.date.accessioned2024-08-31T07:50:05Z
dc.date.available2024-08-31T07:50:05Z
dc.date.issued2024
dc.departmentEge Üniversitesien_US
dc.description.abstractChronic myeloid leukemia (CML) is a clonal myeloproliferative hematological disease characterized by the chimeric breakpoint-cluster region/Abelson kinase1 (BCRen_US
dc.identifier.doi10.1089/jir.2023.0170
dc.identifier.endpage189en_US
dc.identifier.issn1079-9907
dc.identifier.issn1557-7465
dc.identifier.issue4en_US
dc.identifier.pmid38579140en_US
dc.identifier.scopus2-s2.0-85190333642en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage178en_US
dc.identifier.urihttps://doi.org/10.1089/jir.2023.0170
dc.identifier.urihttps://hdl.handle.net/11454/105111
dc.identifier.volume44en_US
dc.identifier.wosWOS:001197190100002en_US
dc.identifier.wosqualityN/Aen_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherMary Ann Liebert, Incen_US
dc.relation.ispartofJournal of Interferon and Cytokine Researchen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.snmz20240831_Uen_US
dc.subjectPonatiniben_US
dc.subjectCombination Therapyen_US
dc.subjectCytokinesen_US
dc.subjectStat5 Inhibitor Pimozideen_US
dc.subjectDrug Resistanceen_US
dc.titlePonatinib and STAT5 Inhibitor Pimozide Combined Synergistic Treatment Applications Potentially Overcome Drug Resistance via Regulating the Cytokine Expressional Network in Chronic Myeloid Leukemia Cellsen_US
dc.typeArticleen_US

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