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    Perlite exposure and 4-year change in lung function
    (Academic Press Inc, 2001) Polatli, M; Erdinc, M; Erdinc, E; Okyay, E
    Perlite is a volcanic glass or amorphous aluminium silicate composed of 71-75% SiO2. When heated to 800-1100 degreesC, it expands to form processed perlite, which has a low density, high surface area, and a low thermal conductivity. The objective was to determine the effect of perlite exposure on pulmonary function tests. Pulmonary function tests in conjunction with chest radiogram were carried out in 36 perlite-exposed workers and 22 unexposed office workers in 1992 and 1996. Respirable dust level exceeded permissible dust levels in work places in the 4 years under study. Transfer coefficient (K-CO) decline was significant in nonsmoker perlite-exposed workers (n = 9), and found to be 5.28 +/- 0.71 (predicted 4.32 +/- 0.11) and 3.84 +/- 0.96 (predicted 4.18 +/- 0.18) 1/min/mmHg, in 1992 and 1996, respectively (P < 0.001). Both smoker perlite workers and office workers showed significant obstruction to airflow in small airways with respect to predicted values and 1-year change in transfer factor (T-L, CO) was significant. Although predicted, 12-year perlite exposure did not lead to a decrease in mean pulmonary function test parameters, there was a tendency to a decline in T-L, CO in the 4-year study period, which may be due to high perlite dust levels. As early effects of perlite dust exposure may not be detected by spirometric measurements alone, the transfer coefficient should be added to spirometry. (C) 2001 Academic Press.
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    Respiratory impairment due to asbestos exposure in brake-lining workers
    (Academic Press Inc Elsevier Science, 2003) Erdinc, M; Erdinc, E; Cok, G; Polatli, M
    There is extensive evidence that exposure to asbestos causes pulmonary parenchymal fibrosis, pleural disease, and malignant neoplasm in asbestos-exposed workers. However, few data concerning brake-lining workers are available in the literature. In this study, we aimed to assess the long-term effects of chrysotile asbestos exposure on lung function and the risk of asbestos-related diseases in brake-lining workers. Seventy-four asbestos-exposed workers who processed brake-lining products and 12 unexposed office workers were offered pulmonary function tests (spirometry and transfer factor) in 1992 and 1999. In 1999, the mean duration of asbestos exposure was 10.00+/-4.07 and 11.02+/-4.81 years (7-31 years) in nonsmoking and smoking asbestos workers, respectively. Transfer factor (T-L, CO) and transfer coefficient (K-CO) decline were significant in the 7-year follow-up in both smoking and nonsmoking asbestos workers. However, lung function indices of the control group, whom were all current smokers; were also found to be decreased, including FEF75, T-L, CO and K-CO. We found minimal reticular changes in 10 asbestos workers who were all current smokers, they underwent high-resolution computed tomography scans of the chest and we found that they had peribronchial thickening resulting from smoking. As a conclusion, even in the absence of radiographic asbestosis, T-L, CO and K-CO may decrease after a mean 10-year duration of exposure to asbestos in brake-lining workers and this is more noticeable with cigarette burden. (C) 2003 Elsevier Science (USA). All rights reserved.
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    Respiratory impairment due to asbestos exposure in brake-lining workers
    (Academic Press Inc Elsevier Science, 2003) Erdinc, M; Erdinc, E; Cok, G; Polatli, M
    There is extensive evidence that exposure to asbestos causes pulmonary parenchymal fibrosis, pleural disease, and malignant neoplasm in asbestos-exposed workers. However, few data concerning brake-lining workers are available in the literature. In this study, we aimed to assess the long-term effects of chrysotile asbestos exposure on lung function and the risk of asbestos-related diseases in brake-lining workers. Seventy-four asbestos-exposed workers who processed brake-lining products and 12 unexposed office workers were offered pulmonary function tests (spirometry and transfer factor) in 1992 and 1999. In 1999, the mean duration of asbestos exposure was 10.00+/-4.07 and 11.02+/-4.81 years (7-31 years) in nonsmoking and smoking asbestos workers, respectively. Transfer factor (T-L, CO) and transfer coefficient (K-CO) decline were significant in the 7-year follow-up in both smoking and nonsmoking asbestos workers. However, lung function indices of the control group, whom were all current smokers; were also found to be decreased, including FEF75, T-L, CO and K-CO. We found minimal reticular changes in 10 asbestos workers who were all current smokers, they underwent high-resolution computed tomography scans of the chest and we found that they had peribronchial thickening resulting from smoking. As a conclusion, even in the absence of radiographic asbestosis, T-L, CO and K-CO may decrease after a mean 10-year duration of exposure to asbestos in brake-lining workers and this is more noticeable with cigarette burden. (C) 2003 Elsevier Science (USA). All rights reserved.
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    Respiratory impairment due to asbestos exposure in brake-lining workers (vol 91, pg 151, 2003)
    (Academic Press Inc Elsevier Science, 2003) Erdinc, W; Erdinc, E; Cok, G; Polatli, M
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    Respiratory impairment due to asbestos exposure in brake-lining workers (vol 91, pg 151, 2003)
    (Academic Press Inc Elsevier Science, 2003) Erdinc, W; Erdinc, E; Cok, G; Polatli, M