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Öğe A case of left atrial myxoma as a cause of platypnea [Platipneye neden olan sol atriyal miksoma olgusu](2006) Tengiz I.; Hamza D.; Türk U.; Ercan E.; Nalbantgil S.[No abstract available]Öğe The importance of plasma B-type natriuretic peptide levels in cardiovascular diseases [Plazma B-tipi natriüretik peptid düzeylerinin kardiyovasküler hastaliklardaki yeri ve önemi](2005) Duygu H.; Türk U.; Zoghi M.; Nalbantgil S.Natriuretic peptide type B (brain natriuretic peptide, BNP) is a neurohormone released by cardiac ventricles in response to volume and pressure load. Recent studies have demonstrated that BNP level measured in the plasma plays an important role in cardiovascular diseases especially in the diagnosis and treatment of heart failure and acute coronary syndrome. This article summarizes the physiology of the brain natriuretic peptide, diagnosis of heart failure and acute coronary syndrome and use of BNP in the treatment and determining the prognosis of cardiovascular diseases.Öğe Myocardial bridgings of the right coronary artery and left anterior descending coronary artery: Very unusual form of myocardial bridge(2005) Duygu H.; Zoghi M.; Kirilmaz B.; Türk U.; Akilli A.[No abstract available]Öğe Statin use is associated with decreased CD-40 ligand expression on T lymphocytes of coronary atheroma plaque in patients with stable coronary artery disease(2008) Türk U.; Alioglu E.; Tengiz I.; Ercan E.; Mahmudov R.; Duygu H.; Türkoglu C.Objective: Atherosclerosis is a chronic inflammatory disease. Statins suppress the inflammation in the plaque. This cross-sectional study was planned to evaluate the effect of statins on plaque T cell activation markers in patients with stable angina pectoris undergoing coronary intervention and atherectomy procedures. Methods: Twenty-six patients with stable angina with suitable for atherectomy coronary lesions were enrolled in the study. Fourteen of 26 patients who had been taking statin treatment for at least six months were assigned to the Group 1 (Statin group) and 12 patients who had not received any lipid lowering treatment comprised the Group 2 (Control group). Atherectomy specimens were studied with single and double immunohistochemical staining (CD25, CD69, and CD40Q. Statistical analysis was performed using Student's t-test and Fisher's exact test. Results: There was no significant difference between the total tissue area of sections (Group 1: 8.4±0.9 mm2, Group 2: 7.8±0.9 mm2, p>0.05). CD3, CD25, CD69, and CD40L positive cells did not show statistically significant difference between the groups in unit area (mm2). There was no significant difference between the groups for percentage of T lymphocytes expressing CD25 (Group 1: 7.8±4.6%, Group 2: 7.8±5.9%, p=0.97) and CID 69 (Group 1: 12.9±4.6%, Group 2: 15.5±5.2%, p=0.203), The expression of CD40L was significantly lower in Group 1 than in Group 2 (Group 1: 4.8±3.9%, Group 2: 11.2±8.7%, p=0.034). Conclusion: We concluded that, statin treatment may decrease the expression of CD40L on plaque T lymphocytes in patients with stable angina pectoris.
