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Yazar "ERSAHIN, Y" seçeneğine göre listele

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    A Case of Rathkes Cleft Cyst Presenting With Diabetes-Insipidus
    (Van Gorcum Co Bv, 1995) ERSAHIN, Y; OZDAMAR, N; DEMIRTAS, E; MUTLUER, S
    Rathke's cleft cysts (RCCs) are considered to arise from the remnants of Rathke's pouch, an invagination of the stomodeum. They are classically described as benign epithelium lined intrasellar cysts containing mucoid material, and also found in 2-33% of routine autopsy series. The most common presenting symptoms are visual impairment, hypothalamic dysfunction, hypopituitarism and headache. Diabetes insipidus has been described in patients with RCC. Very few cases presented with only diabetes insipidus in adults. To our knowledge, our patient is the first case of RCC presenting with only diabetes insipidus in childhood. A 9-year-old girl presented with diabetes insipidus. The physical, neurological and endocrinological examinations were normal, except for diabetes insipidus. Magnetic resonance imaging scan revealed a hyperintense lesion with supra sellar extension in the posterior pituitary both on T1 and T2 weighted images. Subtotal excision of RCC was performed via transsphenoidal surgery. However, diabetes insipidus persisted after the surgery.
  • Küçük Resim Yok
    Öğe
    A Case of Thalamic Hydatid Cyst
    (Van Gorcum Co Bv, 1995) ERSAHIN, Y; MUTLUER, S; DEMIRTAS, E; YURTSEVEN, T
    Cerebral hydatidosis account for approximately 1-2% of patients with hydatid disease. Fifty percent to 75% of intracranial hydatid cysts are seen in children. The cerebral hydatid cysts are usually single and located in the watershed of the middle cerebral artery. To our knowledge, no case of hydatid cyst in the thalamic location has been reported. A 4-year-old boy presented with the left sided weakness. A right thalamic hydatid cyst without rim enhancement and perifocal oedema was detected on the computed tomographic (CT) scan. He was put on albendazole, but headache, nausea and vomiting developed and hemiparesis got worse in the following two weeks. The non-contrast repeat CT showed the pericystic oedema. The rim enhancement and pericystic oedema were also present on magnetic resonance imaging scans. The right thalamic hydatid cyst was removed via the transcallosal approach. The cyst aspiration and intracystic injection of hypertonic saline were performed before the cyst removal. Leakage of the cyst fluid was conceivably the cause of the development of rim enhancement and pericystic oedema. Patients receiving albendazole for the treatment of cerebral hydatid cysts should be closely followed. The surgery is still the choice of treatment in cerebral hydatid cysts, in our opinion.

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