Ersoz G.Ozutemiz O.Akarca U.S.Yilmaz M.Karasu Z.Batur Y.2019-10-272019-10-2719991300-4948https://hdl.handle.net/11454/23535The etiology of gall bladder wall thickening (GBWT) in patients with cirrhosis is unknown. We aimed to compare GBWT in patients with different stages of cirrhosis and to investigate the relationship between GBWT and esophageal varices, ascites, serum albumin and transaminases levels. The patients with liver cirrhosis diagnosed were divided into three groups; from biopsy results group A was composed of 37 patients with no ascites and no esophageal varices; group B, 28 patients with esophageal varices, and no ascites without hypoalbuminemia; group C, 32 patients with esophageal varices and ascites. GBWT was 3.13±0.35 mm (mean±SD) in group A, 3.89±0.83 mm in group B and 5.00±1.39 mm in group C. There were significant differences between group A and B (p<0.0001), group A and C (p<0.0001) and group B and C (p<0.0005). Multiple regression analysis showed that esophageal varices was the only factor related to GBWT (p<0.0001). There was limited correlation between GBWT and ascites (p=0.051) and serum albumin levels (p=0.06). These results suggest that there is a close relationship between GBWT and esophageal varices. GBWT may be considered a sign of esophageal varices and portal hypertension, but a normal gallbladder wall can not rule out esophageal varices.eninfo:eu-repo/semantics/closedAccessCirrhosisGallbladder wallUltrasonographyArticle1011114Q3